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DCP 2.0 (180 Capsules) (New Formula)

Manufactured By: Body Performance Solutions (BPS)

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New & Improved DCP 2.0 by BPS:

Damage Control Protocol!

Body Performance Solutions (BPS) is excited to announce the release of the second generation of the wildly popular fat buner DCP, we are now ready to release to you the newly formulated and super potent DCP 2.0!

While the original DCP worked very well and created quite a large following of raving fans, we did find a couple of limitations based on user feedback and emerging science.

One was directly related to an increase in something called CPT1, which was greatly important for fat burning (and it still is in the new formula) but it ended up depleting the body's carnitine supply which led to side effects such as cramping in some users, and also just plain put a "governer" on the potential rate of fat loss (don't worry, keep reading and this will all be explained).

Not only have we found a way to counter these limitations and thereby eliminating side effects and blowing the doors of the fat burning potential, we've also added several new extremely innovative ingredients while removing TTA (you won't miss it).

The science behind DCP 2.0 is thick, but we've laid it out for you one ingredient at a time in a very digestible format (also with references at the end of each section to make it easier to go look them up), so give it a read and you'll find out right away why we're so excited to bring this revolutionary fat burner to the market.

Artemisia Iwayomogi

Where does it come from?
Artemisia Iwayomogi (AI) is a Korean herb from the Asteraceae family. It is made up of at least 20 known compounds, however it is not known exactly which and how many of these compounds are responsible for providing the numerous benefits.

Current research is pointing towards a couple of the compounds in particular, scopoletin and Dibromo-4-methoxybiphenyl, as having some pronounced benefits in the body, although there are likely many other players at work (1,2).

As we know, herbs such as this are often far more than just the sum of their known parts; for this reason a full spectrum 20:1 ethanol extract has been chosen for inclusion in DCP 2.0.

What is it going to do for me?
AI is an exciting little herb, and although research in humans is in its infancy, the current published data have given us reason to expect big things with regards to accelerating fat loss and improving overall health.

Primary Effects

Activation of PPAR (delta)
PPAR is one of the three main nuclear receptors in the PPAR family (along with alpha and gamma). It can be found in brain, adipose, skin, and skeletal muscle.

Increased expression of this nuclear receptor has been shown to increase fatty acid oxidation in skeletal muscle by shifting the body's metabolic preference away from carbohydrate in favor of lipids (9).

This is favorable for multiple reasons. Obviously this means you'll be burning more fat per unit of time, but one side effect of the body relying less on carbohydrate and more on fatty acids is that the latter is a cleaner burning fuel. During exercise you will produce less lactate, prolonging the accumulation of hydrogen ions as well as reducing the total ROS output. Burn more fat, better workout performance, quicker recovery.

Increased expression of CPT1
AI has been shown to upregulate Carnitine Palmitoyl Transferase (CPT1B), which is a key reason why this herb is going to speed up the fat loss process (1).

CPT1's are a class of mitochondrial enzymes, the "B" subtype are found in skeletal muscle as well as white and brown adipose tissue (WAT & BAT). This enzyme is responsible for transporting long chain fatty acids (LFCA's) across the outer cell membrane so they can be delivered inside the cell to be oxidized. So simply put, more CPT1, more fat is handed to your furnace on a silver platter.

Increased expression of PDK4
Pyruvate Dehydrogenase Kinase Isozyme 4 (PDK4) is an enzyme that phosphorylates something called pyruvate dehydrogenase with the help of ATP, rendering it inactive. This process converts pyruvate to acetyl-coA, thereby increasing energy expenditure. AI has been shown to upregulate this enzyme (1).

Improves efficiency of two out of three steps in the fat burning process
For your body to actually "burn" fat, it has to go through a three step process. Step one is liberating the fat from stored tissue, known as lipolysis. Step two is transporting the fat; step three is actually using it as fuel, known as beta oxidation. AI encourages a higher rate of both transport and oxidation.

Secondary Effects

Intracellular antioxidant, bone growth stimulation
Scopoletin (previously mentioned compound in AI) has been shown to be an intracellular antioxidant, suppressing ROS and superoxide anions in osteoclasts, and looks to be an important player in differentiation of these cells (2).  Research also shows AI can actually stimulate osteoblasts to make new bone (6).

As we learn more about the vital role of bone on regulating full body     metabolism and the importance of osteocalcin in this process through its     interaction on adipokines such as adiponectin, this could provide a metabolic     boost from another pathway (10). We will revisit this when we discuss     Carnitine Fumerate.

Upregulation of secondary fat burning genes
AI has also been shown to upregulate several other genes affecting energy expenditure and lipid efflux including PGC1A and UCP3 (1).

Suppression of inflammatory cytokines
Multiple studies have looked at the effect of mice given a high fat diet with or without concurrent administration of AI. They have found remarkable results including reduction of visceral fat gain, prevention of elevated lipids, leptin, glucose and insulin as well as a reduction in several inflammatory cytokines including TNF-s and IL-6 (1,3).

Hepatoprotective
AI has also been shown in mice to prevent damage from alcohol consumption (yep, they got mice drunk). The mice that weren't lucky enough to get a dose of AI saw their cholesterol and triglycerides jump up and fat burning take a nose dive, while the group that received AI didn't experience these side effects from the alcohol (5).

Allergy relief
One final thing to mention, completely unrelated to fat burning but interesting nonetheless, AI appears to have a positive and significant effect on reducing symptoms of allergies through an interaction with histamine and suppression of inflammatory cytokines (7,8).

Are there any side effects?
No known side effects

Artemisia Iwayomogi References:

1. Cho SY, et al. An ethanol extract of Artemisia iwayomogi activates PPARδ leading to activation of fatty acid oxidation in skeletal muscle. PLoS One. (2012)

2. J Nat Prod. 2013 Mar 19. [Epub ahead of print]
Scopoletin and Scopolin Isolated from Artemisia iwayomogi Suppress Differentiation of Osteoclastic Macrophage RAW 264.7 Cells by Scavenging Reactive Oxygen Species.

3. Evid Based Complement Alternat Med. 2013;2013:915953. doi: 10.1155/2013/915953. Epub 2013 Jan 17.
Artemisia iwayomogi Extract Attenuates High-Fat Diet-Induced Obesity by Decreasing the Expression of Genes Associated with Adipogenesis in Mice.

5.J Food Sci. 2011 Nov-Dec;76(9):T207-11. doi: 10.1111/j.1750-3841.2011.02385.x. Epub 2011 Oct 4.
Comparative study of the hepatoprotective efficacy of Artemisia iwayomogi and Artemisia capillaris on ethanol-administered mice.

6.Biol Pharm Bull. 2010;33(8):1448-53.
Phenolic compounds from Artemisia iwayomogi and their effects on osteoblastic MC3T3-E1 cells.

7.mmunopharmacol Immunotoxicol. 2006;28(3):421-30.
Artemisia iwayomogi inhibits immediate-type allergic reaction and inflammatory cytokine secretion.

8.Exp Biol Med (Maywood). 2005 Jan;230(1):82-8.
Anti-allergic effects of Artemisia iwayomogi on mast cell-mediated allergy model.

9.Diabetologia. 2006 Nov;49(11):2713-22. Epub 2006 Sep 8.
Activation of PPAR-delta in isolated rat skeletal muscle switches fuel preference from glucose to fatty acids.

10.Cell. 2007 Aug 10;130(3):456-69.
Endocrine regulation of energy metabolism by the skeleton.

Catalposide

Where does it come from?
Found in nature in multiple places, Catalposide can be extracted from the unripe fruit of Catalpa ovata as well as the flower Veronica peregrina.

What is it going to do for me?
Catalposide will increase fat loss by one main mechanism

Primary Effects

PPARa (alpha) activation
PPARa activation is directly involved with all three stages of fat burning, and is one of the most important players in lipid metabolism. It is triggered naturally during calorie deprivation and in severe carbohydrate restriction to aid in the production of ketones.

In a 2012 in-vitro study, catalposide treated hepatocytes showed a significant reduction in triglyceride concentrations along with an increased uptake of fatty acids by 70%, an increase in fatty acid oxidation, and suppression of fatty acid synthesis (1).

Secondary Effects

Anti-inflammation
Catalposide has been shown to reduce the inflammatory cytokines in the epithelial cells of the intestines, which could provide a therapeutic effect in those with gut dysfunction (2).

Are there any side effects?
No known side effects.

Catalposide References:

1. Biochem Biophys Res Commun. 2012 Jun 15;422(4):568-72. doi: 10.1016/j.bbrc.2012.05.025. Epub 2012 May 11.
Catalposide is a natural agonistic ligand of peroxisome proliferator-activated receptor-α.

2.Inflamm Bowel Dis. 2004 Sep;10(5):564-72.
Catalposide, a compound isolated from catalpa ovata, attenuates induction of intestinal epithelial proinflammatory gene expression and reduces the severity of trinitrobenzene sulfonic Acid-induced colitis in mice.

Salvia miltiorrhiza

Where does it come from?
Salvia miltiorrhiza is an Asian perennial plant from the Salvia genus.

What is it going to do for me?

Primary Effects

DGAT inhibition
Diglyceride acetyltransferase is the enzyme responsible for the third and final step in producing a triglyceride from glycerol and fatty acids. Inhibit DGAT, which Salvia m. has been shown to do, and you reduce fat accumulation and increase leptin sensitivity significantly (6). If your body wants to store more fat, it cranks up DGAT activity. Not anymore.

Increased glucose sensitivity
Salvia m. has been shown in research to improve uptake of glucose in skeletal muscle, thereby reducing fat storage, preserving lean body mass when dieting, and helping maintain exercise performance (7,8)

Improves lipid metabolism in the liver
When feeding rats a high fat diet for six weeks (which is unnatural for their species unlike humans), the control group showed increased body fat accumulation, hyperinsulinemia, hyperlipidemia, and increased liver enzymes. The group fed a high fat diet in conjunction with Salvia m. did not show any of these negative effects from the high fat diet (9).

Secondary Effects

Cardiac protection
Salvia m. has been shown to inhibit platelet aggregation or blood "stickiness", as well as protecting heart cells from free radicals and inhibit myocardial cell apoptosis (cell death) (1). It has also been shown to protect against LDL cholesterol oxidation (2,3).

Liver and kidney protection
Studies have shown salvia m. to encourage healthy liver and kidney tissues through multiple mechanisms (4,5).

Salvia miltiorrhiza References:

1. Li M, et al. Inhibition of shear-induced platelet aggregation in rat by tetramethylpyrazine and salvianolic acid B. Clin Hemorheol Microcirc. (2004)
2. Zhao BL, et al. Scavenging effects of salvia miltiorrhiza on free radicals and its protection for myocardial mitochondrial membranes from ischemia-reperfusion injury. Biochem Mol Biol Int. (1996)
3. O K, et al. Magnesium tanshinoate B (MTB) inhibits low density lipoprotein oxidation. Life Sci. (2001)

4. Molecules. 2012 Jan 30;17(2):1191-202. doi: 10.3390/molecules17021191.
Protective effect of Salvia miltiorrhiza extract against renal ischemia-reperfusion-induced injury in rats.

5. J Hepatol. 1998 Nov;29(5):760-71.
Salvia miltiorrhiza reduces experimentally-induced hepatic fibrosis in rats.
Wasser S, Ho JM, Ang HK, Tan CE.

6. Arch Pharm Res. 2002 Aug;25(4):446-8.
Inhibitory activity of diacylglycerol acyltransferase by tanshinones from the root of Salvia miltiorrhiza.

7. J Ethnopharmacol. 2010 Apr 21;128(3):575-82. doi: 10.1016/j.jep.2010.01.044. Epub 2010 Feb 1.
Regulation effects on abnormal glucose and lipid metabolism of TZQ-F, a new kind of Traditional Chinese Medicine.

8. Journal of Health Science, 54(2) 203-206 (2008)
Effect of Tangzhiquing on Gluoce and Lipid Metabolism in Genetically Type 2 Diabetes KK-Ay Mice

9. Evid Based Complement Alternat Med. 2013;2013:306738. doi: 10.1155/2013/306738. Epub 2013 Feb 18.
Managing the combination of nonalcoholic Fatty liver disease and metabolic syndrome with chinese herbal extracts in high-fat-diet fed rats.

Dual Carnitine Blend: Propionyl-L-Carnitine (PLCAR) & Carnitine Fumerate

Where does it come from?
Both forms of carnitine are derivatives of the amino acid L-Carnitine found in protein.

What is it going to do for me?

Primary Effects

Resupply carnitine as a transport molecule
Propionyl-L-Carnitine (PLCAR) is included to supply an increased demand for carnitine in the body due to upregulated CPT1 activity from Artemisia. If we crank up CPT1 and don't resupply extra carnitine, which is the main transport molecule for LCFA's, this substrate shortage causes things like cramping and an eventual plateau in fat loss. Of all of the forms of carnitine, PLCAR appears to offer the most benefits to an exercising individual.

Secondary Effects

Osteoblast upregulation
Carnitine Fumerate has been added to DCP 2.0 due not only to its ability to efficiently resupply depleted carnitine in addition to PLCAR, but also because of it's unique ability to positively modulate osteoblast function. This was mentioned previously when discussing Artemisia as having a potentially huge impact on whole body metabolism and energy expenditure, making it the perfect carnitine choice in conjunction with PLCAR (1).

Are there any side effects?
No known side effects, these carnitines will in fact prevent potential side effects of the overall formula (carnitine depletion).

PLCAR & Carnitine Fumerate References:

1. Calcif Tissue Int. 2005 Jun;76(6):458-65. Epub 2005 May 19.
L-carnitine and isovaleryl L-carnitine fumarate positively affect human osteoblast proliferation and differentiation in vitro.

Raspberry Ketones

Where does it come from?
From raspberries, obviously.

What is it going to do for me?

Primary Effects

TRPV1 activation, modulation of norepinephrine and HSL
Activating the Transient Receptor Potential Vanilloid Type 1 protein (TRPV1) offers numerous benefits for fat loss and general health.

Through modulation of NE and hormone sensitive lipase, RK’s appear to be a powerhouse for encouraging fat loss and prevention of fat gain. When mice were fed a high fat diet, RK’s prevented visceral and liver fat accumulation as well as reversing the gain in previously obese mice (3). Recently liver fat has been implicated in metabolic disturbances to an even greater degree than the previous villain, visceral fat, so this is a benefit of great magnitude for overall health (4).

In another study, after 120 days of a TRPV1 agonist researchers concluded adipogenisis and obesity were prevented by activation of TRPV1 channels (5).

Secondary Effects

Pain, anxiety, and depression management (1,2)

Are there any side effects?
No known side effects

Raspberry Ketones References:

1. Starowicz K, Cristino L, Di Marzo V. TRPV1 receptors in the central nervous system: potential for previously unforeseen therapeutic applications. [Internet]. Current pharmaceutical design. 2008 ;14(1):42-54.
2. Gibson HE, Edwards JG, Page RS, Van Hook MJ, Kauer JA. TRPV1 channels mediate long-term depression at synapses on hippocampal interneurons. [Internet]. Neuron. 2008 ;57(5):746-59.
3. Morimoto C, Satoh Y, Hara M, Inoue S, Tsujita T, Okuda H. Anti-obese action of raspberry ketone. [Internet]. Life sciences. 2005 ;77(2):194-204.

4. Fabbrini E, Magkos F, Mohammed BS, Pietka T, Abumrad NA, Patterson BW, et al. Intrahepatic fat, not visceral fat, is linked with metabolic complications of obesity. [Internet]. Proceedings of the National Academy of Sciences of the United States of America. 2009 ;106(36):15430-5.

5. Zhang LL, Yan Liu D, Ma LQ, Luo ZD, Cao TB, Zhong J, et al. Activation of transient receptor potential vanilloid type-1 channel prevents adipogenesis and obesity. [Internet]. Circulation research. 2007 ;100(7):1063-70.

Mangiferin

Where does it come from?
Mangiferin is known as a xanthanoid, and it is found in mangoes as well as a few other places in nature.

What is it going to do for me?

Primary Effects

Increased expression of PPAR-a
This is the second PPARa activator in addition to catalposide, magniferin has been shown in several studies to cause an effective upregulation (1,3,4). As a refresher on PPARa, its activation is directly involved with all three stages of fat burning, and is one of the most important players in lipid metabolism. It is triggered naturally during calorie deprivation and in severe carbohydrate restriction to aid in the production of ketones.

Decreased expression of PPAR-gamma
PPAR-gamma is responsible for increasing storage of fat in the fat cell, mangiferin has been shown to reduce its activity thereby reducing fat storage (2).

Downregulation of DGAT
This is the second compound in the formula to target DGAT in addition to Salvia m. As a refresher, diglyceride acetyltransferase (DGAT) is the enzyme responsible for the third and final step in producing a triglyceride from glycerol and fatty acids. Downgregulate DGAT, which magniferin has been shown to do, and you reduce fat accumulation and increase leptin sensitivity significantly (1).

Increased expression of CPT1
Along with Artemisia, this is the second ingredient in the formula to target CPT1. As a refresher, CPT1's are a class of mitochondrial enzymes, the "B" subtype are found in skeletal muscle as well as white and brown adipose tissue (WAT & BAT). This enzyme is responsible for transporting long chain fatty acids (LFCA's) across the outer cell membrane so they can be delivered inside the cell to be oxidized. So simply put, more CPT1, more fat is handed to your furnace on a silver platter. Mangiferin has been shown to effectively upregulate CPT1 (1).

Increases Lipoprotein Lipase (LPL)
A 2011 study demonstrated mangiferin's ability to crank up LPL levels, a necessary enzyme in the fat burning process (1).

Improves glucose utilization
This is accomplished by increasing GLUT4 density on the muscle cell (where insulin binds to allow glucose to enter) as well as activating AMPK (an important cellular energy signal) (2,5).

A brand new study done in 2013 shows mangiferin reduced cognitive decline associated with the downstream effects of diabetes, by suppressing methylglyoxal hyperactivity. This reduced advanced glycation end products, oxidative stress and inflammation (6).

Secondary Effects

Prevention of differentiation of adipocytes
Satellite cells are like babies that can grow up to be whatever they want to be. Or, more accurately, whatever they're told to be. If a satellite cell is informed that calories are in excess, and the host needs more silos to store all the grain (metaphor intended), these cells are instructed to become adipocytes. However this doesn't have to be the case, these cells could grow up to be neurosurgeons, firefighters, or myocytes. Mangiferin helps steer these impressionable young cells in the direction you want, more muscle, less fat.

Are there any side effects?
Carnitine depletion.

Mangiferin References:

1. Mol Nutr Food Res. 2011 Dec;55(12):1809-18. doi: 10.1002/mnfr.201100392. Epub 2011 Oct 31.
Beneficial effects of mangiferin on hyperlipidemia in high-fat-fed hamsters.
Guo F, Huang C, Liao X, Wang Y, He Y, Feng R, Li Y, Sun C.

2.Clin Nutr. 2009 Oct;28(5):565-74. doi: 10.1016/j.clnu.2009.04.018. Epub 2009 May 23.
Salacia oblonga extract increases glucose transporter 4-mediated glucose uptake in L6 rat myotubes: role of mangiferin.
Girón MD, Sevillano N, Salto R, Haidour A, Manzano M, Jiménez ML, Rueda R, López-Pedrosa JM.

3. Life Sci. 2008 May 23;82(21-22):1045-9. doi: 10.1016/j.lfs.2008.03.005. Epub 2008 Mar 28.
Salacia root, a unique Ayurvedic medicine, meets multiple targets in diabetes and obesity.
Li Y, Huang TH, Yamahara J.

4. Toxicol Appl Pharmacol. 2006 Feb 1;210(3):225-35. Epub 2005 Jun 21.
Salacia oblonga root improves postprandial hyperlipidemia and hepatic steatosis in Zucker diabetic fatty rats: activation of PPAR-alpha.
Huang TH, Peng G, Li GQ, Yamahara J, Roufogalis BD, Li Y.

5.Pharmacogn Mag. 2013 Jan;9(33):72-5. doi: 10.4103/0973-1296.108145.
Effect of Mangiferin and Mahanimbine on Glucose Utilization in 3T3-L1 cells.
Kumar BD, Krishnakumar K, Jaganathan SK, Mandal M.

6.Psychopharmacology (Berl). 2013 Mar 26. [Epub ahead of print]
Suppression of methylglyoxal hyperactivity by mangiferin can prevent diabetes-associated cognitive decline in rats.
Liu YW, Zhu X, Yang QQ, Lu Q, Wang JY, Li HP, Wei YQ, Yin JL, Yin XX.

Momordin

Where does it come from?
Momordin is a bioactive glycoside extracted from the bitter melon fruit grown throughout Asia, Africa & The Caribbean.

What is it going to do for me?

Primary Effects

PPAR (delta) activator
This is the second ingredient in the formula to target PPAR?, alongside Artemisia.

As a refresher, increased expression of this nuclear receptor has been shown to increase fatty acid oxidation in skeletal muscle by shifting the body's metabolic preference away from carbohydrate in favor of lipids (9).  Momordin has been shown to upregulate human PPARdelta expression in vitro (1).

Inhibit fat storage and increase lipolysis
A study done in 2011 showed Bitter Melon Juice (containing momordin) was able to have a two-pronged attack on fat metabolism by inhibiting its storage as well as increasing rate of lipolysis in human fat cells (2).

Improve blood glucose and insulin sensitivity
In mice fed a high fat diet, the group receiving Bitter Melon bioactives lost weight, improved gluose metabolism and raised insulin sensitivity by increasing GLUT-4 density in skeletal muscle cells (3). It can also potentially slow gastric emptying, which improves glucose metabolism and insulin signaling (7).

Secondary Effects

Being looked at for anti-cancer potential (4,5)

Hepatoprotective
Momordin has been shown to enhance the liver's antioxidant systems (6).

Anti inflammatory/pain reducer
In animals, momordin has been shown to reduce sensitivity to pain through an anti-inflammatory mechanism (8).

Are there any side effects?
No known side effects

Momordin References:

1. J Atheroscler Thromb. 2009;16(6):888-92. Epub 2009 Dec 22.
Activating effect of momordin, extract of bitter melon (Momordica Charantia L.), on the promoter of human PPARdelta.

2. Momordica charantia (bitter melon) inhibits primary human adipocyte differentiation by modulating adipogenic genes. The Journal of nutritional biochemistry 2011

3. Bioactives from bitter melon enhance insulin signaling and modulate acyl carnitine content in skeletal muscle in high-fat diet-fed mice.

4. Apoptosis. 2013 Jun;18(6):751-65. doi: 10.1007/s10495-013-0820-z.
Momordin Ic induces HepG2 cell apoptosis through MAPK and PI3K/Akt-mediated mitochondrial pathways.
5. Induction of apoptosis by momordin I in promyelocytic leukemia (HL-60) cells.

6. J Med Food. 2005 Summer;8(2):177-83.
Momordin Ic and oleanolic acid from Kochiae Fructus reduce carbon tetrachloride-induced hepatotoxicity in rats.
7. J Pharmacol Exp Ther. 1999 May;289(2):729-34.
Inhibition of gastric emptying by triterpene saponin, momordin Ic, in mice: roles of blood glucose, capsaicin-sensitive sensory nerves, and central nervous system.

8. Biol Pharm Bull. 1997 Oct;20(10):1086-91.
Studies on kochiae fructus. III. Antinociceptive and antiinflammatory effects of 70% ethanol extract and its component, momordin Ic from dried fruits of Kochia scoparia L.

9.Diabetologia. 2006 Nov;49(11):2713-22. Epub 2006 Sep 8.
Activation of PPAR-delta in isolated rat skeletal muscle switches fuel preference from glucose to fatty acids.

These statements have not been evaluated by the Food and Drug Administration. This product is not intended to diagnose, treat, cure or prevent any disease.


Customer Reviews for DCP 2.0 (180 Capsules) (New Formula) by Body Performance Solutions (BPS)

Write a review and share your thoughts with other customers.

  • Currently 5/5
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Too many good! By: Touey

Review posted on: 2013-12-05 00:49:36 -0500

If you do know what which you should be trying this as best for stubborn fat. Best with alphamine or Alpha t2 ... enjoy too many losses! thanks to good weight loss for ever.

  • Currently 5/5
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unbelieveable product!! By: Christopher

Review posted on: 2013-07-22 13:49:39 -0400

Let me just say this is probably one of the best supps I've ever run, and definitely the best of its kind. I've been on a consistent calorie surplus with some very high carb days that would typically leave me bloated. I found that by taking 2 caps AM, 2 caps PM and 4 at bed time, if I ate carbs late at night, instead of waking up boated and soft, my muscles felt tighter and harder and I had more power behind my lifts. I think the nutrient optimizing qualities of DCP2.0 also helped my recovery out a lot allowing me to lift heavier, and hit PRs almost every single session! I can only imagine how awesome this stuff would be with a recomp or cut protocol to help optimize nutrient utilization and recovery. Overall, I give it a 10/10 despite the weird tasting burps

  • Currently 5/5
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Best Non Stimulant Fat Burner By: Josh

Review posted on: 2013-07-07 19:30:17 -0400

I stack lean xtreme with this and after 1 bottle of each in 30 days im ripped! Its a top notch formula for sure. Look no further add this to your fat burning stack trust me.

DCP 2.0 Supplement Facts

Serving Size: 2 capsules
Servings per Container: 90

   
  Amount Per Serving % Daily Value
Salvia Miltiorrhiza 20:1 250 mg **
Artemisia Iwayomogi 20:1 500 mg **
Catalposide 150 mg **
Propionyl-L-Carnitine (PLCAR)/Carnitine Fumerate Blend 300 mg **
Raspberry Ketones 100 mg **
Mangiferin 50 mg **
Momordin 35% 40 mg **
 

* Percent Daily Values are based on a 2000 calorie diet.
** Percent Daily Values not established.

INGREDIENTS: Magnesium Stearate, Rice Flour, FD&C Red# 40, FD&C Blue# 1.

DCP 2.0 Directions:

Take 2 capsules 3 times daily as needed or directed by your physician.

DCP 2.0 WARNING:

Consult your physician before using this or any dietary supplement. Do not take if you are pregnant or breast feeding, elderly, chronically ill, have difficulty urinating due to prostate enlargement, or taking any prescription or over-the-counter medicine, including but not limited to antidepressants (such as MAO inhibitors), allergy medications, and medications for high blood pressure or other cardiovascular conditions. Discontinue use and contact your doctor if you experience dizziness, headache, nausea, or heart palpitations. KEEP OUT OF REACH OF CHILDREN.